Dihydrotestosterone (DHT) is a sex steroid and androgen hormone. In the body, the 5a-reductase enzyme synthesizes DHT in the adrenal glands, testes, hair follicles and prostate, giving males man-like features. In males, 5% of testosterone that is produced gets converted to DHT, so anytime testosterone is increased, you will also increase DHT. DHT does not convert to estrogen. Since Masteron is a DHT derived compound, you can expect much more increased DHT in the body, without estrogen increasing. Masteron carries relatively low anabolic and androgenic ratings; however, these ratings are somewhat misleading. It’s important to remember DHT, the basis of Masteron, is five times more androgenic than testosterone with a much stronger binding affinity to the androgen receptor. This again promotes a harder look and can also enhance fat loss. Most all anabolic steroids are well-noted for enhancing the metabolic rate, but strong androgens have a tendency to directly promote lipolysis.
Because the ultimate goal of a steroid cycle is to increase strength and muscle size, the associated spike in estrogen which accompanies steroids such as Testosterone is considered undesirable. In order to disassociate the two effects, two classes of drug are used. Medications such as Nolvadex or Clomid target the estrogen receptors. They make it more difficult for the estrogen to exert it’s influence within the body thus allowing the testosterone to act more freely. The second class is aromatase inhibitors such as Femara. They target the aromatase enzyme itself in order to prevent the production of estrogen in the first place. Sometimes, it’s not always clear which option you should go with or even what the differences are between the two. Lets clear that up a little.